(upbeat music)
- Welcome to the Critical Care Obstetrics Podcast.
I'm Stephanie Martin, Medical Director
at Clinical Concepts in Obstetrics,
and I'm here today with my partner,
Suzanne McMurtry-Baird,
Nursing Director at Clinical Concepts in Obstetrics.
Today, we're following up on our series
on vital signs are vital.
We've already covered three vital signs,
heart rate, blood pressure,
and the ever-missing respiratory rate.
And today, we're gonna continue our conversation
about the lungs and respiration
and expand it to oxygenation
and focus on the oxygen saturation.
So hopefully, you guys are learning a lot from this,
but if you wanna learn more
about high-risk and critical care obstetrics,
you should check out our Critical Care OB Academy
that's available online at our website,
clinicalconceptsinob.com.
And I wanna also highlight
that we've been getting a lot of requests
for group pricing.
Maybe you wanna explore if many nurses
on your unit can take it
or you're in a residency training program
and you wanna have your residency class take it.
We are more than happy to provide group pricing,
so please reach out through the website.
And we'd love to hear more feedback
about what you guys think about
the online Critical Care OB Academy.
So let's dig into oxygen saturation.
As always, before we get into details
about the oxygen saturation,
we wanna highlight a few key points
about vital signs in general.
These are crucial points to remember.
You've heard me say before
that if you wanna annoy me, call me with one vital sign.
I wanna know all the vital signs,
so please do a full recording of the vital signs.
A single vital sign measurement
really is not very good at predicting adverse events.
You need combinations,
the pulse plus the heart rate,
the pulse plus the blood pressure,
plus the respiratory rate, et cetera.
And these combinations really improve your ability
to understand what's happening
and predict whether bad things are on the way.
The more you understand vital signs,
the more uncomfortable you will be
with a single vital sign,
and the more uncomfortable you will be
when you identify one as abnormal.
It's really also not just about a single vital sign,
more than just the combinations,
but also trends over time.
Like what's actually happening with your patient?
- Yeah, I was thinking about that, Stephanie,
when we were talking in the last episode
about the early warning criteria,
many of the algorithms and the early warning criteria
are combinations of vital signs.
So it's not just one standalone
and they need to be sustained.
So obviously you don't wanna watch
for too long of a period of time,
but let's say you do a pulse oximetry
and you're watching that trend,
or you take a respiratory rate and it's too high,
then where has that patient been?
So it's a combination of those two
or something else in combination
to look at that early warning criteria.
- Absolutely.
It's so, so important to understand
what's happening with those vital signs,
and you really can't understand that
with a single vital sign
that's not repeated over time
and in isolation without combining it with other things.
So before we talk about hypoxemia
and oxygen saturation in general,
I wanna talk about the equipment.
So I'm gonna keep it real simple.
I go into it in a lot more detail in the academy,
but very, very simply,
when you put a pulse oximeter on someone's finger
or their earlobe or their toe or whatever,
you're essentially shining red light through the tissue.
And the computers are calculating
how much light is absorbed
and that gets translated into how red the blood is
in very basic terms, how red is the blood.
And the redder the blood,
the more saturated hemoglobin is with oxygen molecules.
So it's very simply looking at,
is the blood this red or less red?
More red or less red?
And the computers have done the calculations for you
and they translate that to the percentage of hemoglobin
that is saturated or carrying an oxygen molecule.
So in order for the equipment to work,
you have to have a good pulse.
You've got to have blood flowing through the digit
or through whatever.
- Yes, that's such a good point.
I remember you and I were in a room one time
and we had this patient that was hemorrhaging
and we had trained this team.
We had trained them and the first thing I saw
was a nurse coming out of the room during this emergency.
And I said, "Where are you going?"
He said, "Well, the pulse oximetry is not working."
And I was like, palpator pulse,
like hands on palpator pulse.
And you could see the nurse palpating
and not getting or really feeling a strong quality pulse
or it being absent.
And I thought, yep, yes,
it's different hearing it in a classroom or on a podcast
or in a lecture, but really in those clinical situations
and you're not getting away from,
it's not the equipment.
It's the fact that this patient has hemorrhaged so much
that she doesn't have a quality pulse
and the quality is so weak
that you're not getting able to get away from.
So I just wanted to put that in place
because even with all the training
and us talking about it now,
I want you to think about that the next time
you have a patient that you're not getting away from on
or you start to see it being dampened
or intermittent readings,
how pay their pulse 'cause I love pulse quality.
- We love a strong pulse.
- I love a strong pulse.
- Always assume your equipment is working.
I mean, think yourself,
you've probably changed out the pulse oximeter attachment,
the sticky pads,
that portion of the device,
countless times in your career,
but did it ever really fix the problem?
Was it ever really the problem?
Okay, well, maybe if all the sticky was worn off
and had been taken off a million times
and it's not holding in place or whatever,
but the reality is most of the time
the equipment's working just fine
and it's us who's not appreciating
that that fingertip is not getting an adequate perfusion
of blood because your patient is running out of blood.
- Right.
- Or she's in septic shock
and the body doesn't think
that the fingertip is an important part.
It's gonna be perfusing kidneys and brain.
So just assume your equipment's working, okay?
- Right.
And that's easy to shoot, you know, troubleshoot, you know?
- Real easy.
Just see if it's fixing, you know,
if it's attached well where it needs to be
and check their pulses.
So let's talk about hypoxemia.
Now, commonly we interchange hypoxemia and hypoxia.
They are actually not the same thing.
I'm sure I have misspoken on many an occasion,
but hypoxemia is low oxygen level in the blood.
If left unchecked and uncorrected,
it will ultimately lead to hypoxia,
which is low oxygen levels in the tissues.
And that's what we're trying to avoid by intervening early.
We wanna correct the amount of oxygen in the blood
so that we can improve the likelihood
we're gonna have good amount of oxygen in the tissues.
So hypoxemia in obstetrics is defined
as a hemoglobin saturation or an oxygen saturation,
pulse ox, O2 sat, whatever you wanna call it,
of less than 95%.
In a pregnant patient, your pulse ox should be,
your oxygen saturation should be 95% or greater.
And it's important to understand,
like we've been highlighting,
we're not looking at these values in isolation.
You wanna see trends over time.
It's not unusual to have a patient sat,
drop down, all of a sudden it's 88, 89
while she's eating lunch.
And then as soon as she holds her hand still,
you get it attached well,
you make sure you've got a good connection.
We're back in the 99 to 100 that she's been all day long.
So a single value is really not useful in isolation.
You need to be looking over time.
- Yeah, and that's, I remember that came up,
oh, it comes up at the bedside all the time.
I remember having a nursing student with me one time
and said, her pulse oximeter value keeps jumping down.
And then it goes back to normal and it jumps down
and goes back to normal.
I said, because she's moving
and you're getting an erroneous value in there.
Which I think is also important to bring up
because again, those values come over to your machine.
So if you have like a little disconnect
in your pulse oximeter sticky
that we talked about earlier,
or if she's moving a lot,
then you're gonna get some erroneous values
and they automatically go over into your documentation.
You need to state something like,
you know, obviously if her pulse oximeter value was,
you know, 78 truly, then she would be in a crisis, right?
So, and then it's gonna come right back up to 98%.
That's not the way this technology works, right?
So oxygen doesn't hop on to hemoglobin
and hop off that fast and then reload immediately.
So there's some kind of issue
if she were to sustain a pulse oximetry value around 78.
But to have an erroneous value here and there,
that's just movement.
- Now the other, like the opposite of this
where some of these values might actually be real
and critical.
So what we're describing are erroneous measurements,
which are just quality of your measurement typically, okay?
And you guys can tell that easily.
You all know it when it happens.
You see it, it jumps down and comes right back up
with just some little fiddling or whatever.
But if you have a truly critically ill patient,
they might be on the edge
and they might not have a lot of oxygen dissolved
in their blood.
And so just rolling them over in the bed
or turning them on their side will cause them to desaturate.
And that is a consumption issue
and an oxygen delivery issue in a critically ill patient.
We talk a ton more about this in the Academy.
But again, it's not gonna go down in a blink
and come back up in a blink.
You'll see it go down and then come back up
as they get back into a resting position
and their demand and their consumption goes down
and their delivery improves.
So there are times where those changes
are actually real and important,
but that's where your trends and your combination
with other vital signs will help you understand
what's happening.
So let's talk about what the saturation actually means.
So when we say oxygen saturation,
the other way to think of it is hemoglobin saturation
because each hemoglobin molecule
can carry a fixed amount of oxygen.
So hemoglobin as a molecule
can carry a certain amount of oxygen
and it can't carry any more than that.
You can't overload it.
It's gonna carry as much as it can
and that's 100% saturated.
Then as it delivers oxygen to your tissues,
it releases the oxygen molecule
and then it is less saturated than it was before.
So if you look at venous blood,
'cause everything we're talking about right now is arterial,
if you look at venous blood,
it's not unusual for it to be in the 70s and 80s.
That's what you want it to be
because that means that oxygen has been delivered to tissues
and it's ready to be reloaded again.
So the saturation tells you is the hemoglobin molecule
holding as much as it can hold.
And that's what that 95% or greater tells you.
No one is 100% all the time.
That's just not realistic.
We just want you to be good enough, 95% or greater.
Now in the non-obstetric population,
those of you who work in ICU's and med surge floors,
you have a different threshold for quote, okay,
and that's 92% or above.
And the big difference is the fetus
because the fetus has to have oxygen delivered as well.
And if you are on the,
so remember your oxy hemoglobin dissociation curve
where you've got the balance between
how much oxygen is in the blood
and how much oxygen is on your hemoglobin molecules,
the lower your saturation gets,
then the more rapidly your patient
is going to deteriorate clinically
and have a rapid drop in their oxygen saturations.
So you don't want them living on that cliff
if they've got a baby that they've also got to support.
Plus pregnant women have a much higher metabolic need,
et cetera, than other patients.
So we set a threshold of 95 and above.
If they're not pregnant,
we generally will accept a saturation of 92 and above.
- Yeah, I want to highlight that a little bit,
'cause when we go into hospitals,
sometimes that is in the physician orders
for obstetrics as well as highlighted as abnormal,
not highlighted, in other words,
at that particular hospital until it reaches 92%.
And when we have a patient that reaches 92%,
that means their PAO2 is much lower
at that particular point in time.
And it's like you said, they're on that cliff
and they're ready to dive off.
And you'll start seeing fetal changes,
which is the only evidence that we have
that shows that oxygen improves fetal outcomes
is when maternal PAO2 is low
and we are trying to increase the PAO2
as well as increase their oxygen content
as well as their oxygen delivery.
So, and that's the rationale for MASCO2
in a pregnant woman.
And the only evidence that we have
that it's beneficial to the fetus
is when mom's saturation drops or when her PAO2 drops.
So just to highlight on that,
because I know that that topic comes up,
which will probably be another podcast in the future
because lots of evidence keeps coming out
and a lot of controversy over that topic.
- So, couldn't agree more.
Let's talk about PAO2 real quick or PAO2, same thing.
So the saturation is how much oxygen
that the hemoglobin itself is carrying.
The PAO2 or the PAO2 is all the leftover oxygen
that gets delivered to the bloodstream.
Once the hemoglobin can't carry anymore,
it's 100% saturated,
all the extra gets dissolved in the blood.
Okay, just think of it as a highly carbonated drink.
The more oxygen you put in,
the more gets dissolved in the blood.
And so it's just sitting there ready to fill hemoglobin
as it delivers oxygen.
So when you have a decrease in your oxygen saturations,
when you have high poxemia,
that essentially means that she does not have
enough oxygen dissolved in the blood
to keep hemoglobin full.
That is abnormal.
You should always be able to fill the hemoglobin tank
and just load it up with oxygen to its maximum capacity.
You're not gonna have a high level
of oxygen dissolved in the blood
and a low oxygen saturation in a normal situation.
The exception to that is carbon monoxide poisoning.
But we're talking about a normal situation.
You're not gonna have low saturations and high PO2s.
Your PO2, your dissolved oxygen is gonna get all used up
in order to keep the hemoglobin fully saturated.
- That's the reason why we don't draw
a lot of arterial blood gases anymore.
We used to draw a lot more than we do now
with the technology and the understanding of post-oxymetry.
And so unless you have a patient on a ventilator
or an acute compromise,
you may or may not do an arterial blood gas,
at least not at the frequency that we used to.
- Yeah, the gases are super, super helpful
when you're giving the patients getting a lot of oxygen
and you don't know how much is getting in.
So that's 'cause you can saturate your hemoglobin,
but how much is actually getting through
and dissolving in the blood?
So we don't just routinely do them
just because they have a low sat.
If they've got a low sat, I know what their PO2 is.
But when I'm giving 'em, let's say they're intubated,
they're on 100% oxygen and their sats are 96, 97,
I have no idea how much oxygen is dissolved in the blood
until I check it.
And so there's a role for it, but I totally agree.
We're not just doing ABGs left and right
just because we wanna know the PO2.
I can guess it.
- Yeah, I remember, I practiced at a time
when we didn't have post-oxymetry.
And I think that again,
understanding the technology and what it even means
is so helpful because if we were never really taught that
when it first came out, we were taught,
hey, put this on their finger or their ear.
And I remember the little rubber things
that fit on the finger that everybody used,
which kinda grosses me out when I think about it now.
But yeah, but we didn't really understand the technology.
And I don't think that we've really taught it
to all the clinicians that utilize this technology
on what it even means.
So I hope this has been being helpful
to understand some of the ins and outs.
- So now let's dig into the why
because we think it's crucial that you understand the why.
So causes, why does somebody have
a decreased oxygen saturation?
Well, first of all, if you're practicing at altitude,
like I did for eight years of my career,
when you're breathing in lower amounts of oxygen,
you're gonna have lower amounts of oxygen in your blood.
And there's no exception during pregnancy.
So the higher altitude you are,
then the lower amount of oxygen you're breathing in,
but those patients should still be able
to get their saturations up to 100%.
They just might live at a little bit lower level.
But generally speaking, they're adapted
and everyone does well.
I'm not gonna talk about high altitude medicine
at this point, but you breathe in less oxygen,
you're gonna have lower levels of oxygen in your blood.
- I feel it when I go to Colorado.
- That's why they sell the oxygen in cans
and you put the little can over your face
and just breathe it in and yeah.
- You go to those little oxygen bars or something, you know?
- Yes, they flavor it.
- They can, yeah, I know, I've seen flavors.
- So if you're paying for extra oxygen at the oxygen bar
and you're in Las Vegas at sea level,
all you're doing is raising your PO2,
you're not doing anything else.
- I've done that, have you done it?
Were you with me or did you do that with me?
- No, I didn't do it.
- Okay, I did it.
- But I was there.
What happens in Vegas stays in Vegas.
Who's Anne, stop talking.
- Oh yeah, that's right, that's right, I'm sorry.
I'm really being distracting today.
- So otherwise, understand the why.
So respiratory compromise, this is the number one thing
that we are worried about.
It's usually the reason we're monitoring our patients.
We wanna know what's happening
with their respiratory system.
Are they compromised?
Are they oxygenating well?
And in a pregnant patient,
when you have a patient who's hypoxemic,
you're really talking about one of four things,
the overwhelming majority of the time.
And that's pulmonary edema,
that can be cardiogenic or non-cardiogenic,
that's a different podcast.
And we talk about all of these things a ton in the academy.
It can be asthma, asthma's just common, right?
Pneumonia of any kind, community acquired,
aspiration, COVID, whatever.
And pulmonary embolism, a blood clot.
Now they all cause decreases in oxygen levels
for different reasons,
but the final result is gonna be hypoxemia
and then ultimately low PO2 levels if it's not corrected.
So decreased oxygen in the air like at altitude,
respiratory compromise.
- And those are gonna present totally different too.
So again, assessment goes along with this,
not just a value of a pulse oximeter.
- Yep.
And then next is increased utilization.
So when you're using a ton of oxygen,
sometimes you just can't keep up.
Like patients that are very febrile or septic
or in septic shock,
that might be a combination of pulmonary edema
in conjunction with increased utilization
because their metabolic rate is higher,
they're fighting infection, whatever it may be.
And so a lot of these things
are in combination with each other.
It's not just one issue that's causing it.
And then the last one to think about is lack of perfusion
like we talked about before.
If you're not perfusing that tissue,
you're gonna have a decreased pulse pressure,
you're gonna have wave form issues,
you may not be able to register the saturations.
And the two most common scenarios
where we'll see that in an obstetric population
is gonna be sepsis and hypovolemic states like hemorrhage.
So decreased inspired oxygen, respiratory compromised,
increased utilization, lack of perfusion
or a combination of all of the above.
You can absolutely have a patient who's septic,
who's not perfusing,
who's got increased utilization
and has non-cardiogenic pulmonary edema
and maybe even pneumonia on top of it.
So lots of reasons why they could have low oxygen
and you're gonna be addressing each one in a different way.
I know that, and fever, if you add that to it,
think about how the utilization occurs in that scenario
and how you can drop your oxygen saturation
just from that ton of utilization,
much less if you had respiratory compromise on top of it.
- Absolutely.
And then I'm gonna close out
with a last little pearl to remember.
- Your favorite.
Anemia does not cause hypoxemia.
So don't ever blame low oxygen saturations on anemia.
It's not a thing.
You can have one gram of hemoglobin
and it can still be 100% saturated.
All the sat tells us is how much oxygen
is that hemoglobin molecule carrying?
Is it carrying its max amount
or is it not carrying the max amount?
Whether you have one gram or 14 grams, okay?
Now that's different than oxygen content.
Your body has to have enough hemoglobin molecules
to deliver enough oxygen to all your tissues,
but that doesn't mean that it's not saturated.
That's a completely different thing.
Suzanne, you're nodding your head.
- Yeah, well, because how I love to teach that concept
is again, this is just a piece of technology
that gives us a ton of information,
but you have to also look at it
in what's going on with the patient.
And if you don't know her hemoglobin,
then you really can't estimate
how much oxygen content that the patient has
without that value.
So like you said, you could have one,
you could have five grams of hemoglobin
and it'd be 100% saturated,
but she still doesn't have enough oxygen content.
Therefore, she's not gonna have adequate oxygen delivery
to the tissues and her tissues can still be hypoxic
because she doesn't have enough delivery
and that is due to the content,
but still may be 100% saturated.
So one piece of technology does not replace respiratory rate
as well as your other clinical assessments
that go along with it.
- And I talk about all of those concepts
and more in the Academy.
I have a whole section on oxygen delivery
and content and consumption, et cetera,
and give some clinical examples there.
So if you wanna learn more about this
or many other important concepts
and high risk and critical care obstetrics,
you can find our Academy online
at clinicalconceptsinob.com.
We're so grateful to you guys for listening
and for supporting our podcast.
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