Leading Cancer Researcher: They’re Ignoring My Research, Cancer Patients Must Know This!

Perustin pyöräilyvaatteita valmistavan ornotin vuonna 2013. Mielestäni parasta Shopify saan se, että voimme harjoittaa liiketoimintaa ilman teknistä osaamista. Voimme hallinoida yrityksen taustajaristeelmiä ja frontendia sekä myydä verkossa, vaivatta. Jos Shopify olisi pyöräilyvaruste, sollisimielestäni itse polkupyörä. Sillä asiat hoidetaan ja meidän liiketoiminta me hoituu Shopify'ssa. Aloita ilmainen kokeilu, Shopify piste komsi vustolla. Sitten on enää kokeilu, ei ole kokeilu, se on kokeilu. Sitten on enää kokeilu. Sitten on on, että parasta piste kokeilu, koska kokeilu on todella muuta. Ja itse on kokeilu, tarvitaan ja seurantajaa. Se on kokeilu, että se on kokeilu. Ja tekee minä tämän kokeilu, joten on joten kokeilu. Se on kokeilu, joten on kokeilu. Ja tekee kokeilu, on kokeilu, on kokeilu, on kokeilu, joten on kokeilu. Ja, mutta se on kokeilu, joten on kokeilu, kokeilu, kokeilu. Se on kokeilu, joten on kokeilu, joten on kokeilu, joten on kokeilu, joten on kokeilu, joten on kokeilu. Ja se on kokeilu, mutta kokeilu, mutta kokeilu, joten on kokeilu, joten on kokeilu. Ja tekee minä tämän kokeilu. Ja se on kokeilu. Ja se on kokeilu, joten on kokeilu, joten on kokeilu. Ja se on kokeilu, joten on kokeilu. Ja se on kokeilu. kun lähteä mu pursei on aika laju. On makimosiita muista, mitä sientoa hienotu robbedtiin ajatsksie Usalli, mutta se on significanceun funcionus. Sewisista yhteencipatna ei liuéllisen edelä m Hyunickedia gustoja na kommen! Meidnakkoalaa olen ma circuits ainakin on hyväsi». Vi shoutedannan muista saasiedon keskilla, joten on katsomassa, ja minkäkoa on katsomassa, ja minkäkoa on katsomassa? Ja mä oon tämän katsomassa, että se on katsomassa. Ja mä oon katsomassa, ja mä oon katsomassa, ja mä oon katsomassa, ja mä oon katsomassa, -What is it that you've committed your life to doing, Thomas? -Well, we're right now committed our lives to managing cancer effectively, without toxicity, which is based on the science that I and others have done in this field. -You have a perspective on treating cancer and other metabolic diseases that others don't have. The mainstream should I say? -Oh, yeah. Well, mainstream doesn't have it for sure. But it's based on science. I mean, my work is based on what Otto Werberg, the famous German scientist, said from the 1920s, 30s and 40s. He clearly showed that cancer was a mitochondrial metabolic disease. -What does that mean, mitochondrial metabolic disease? -Okay. It means that the origin of the disease resides in the organelle called the mitochondrion. It's in the cytoplasm of the cell. It used to be called, still is the powerhouse of the cell, it gives the cell the energy. -I think we have a mitochondrion. What am I team bringing a mitochondrion? -Well, you have a mitochondrion here. -Oh, here we go. -But one each here. -Well, see, this is the little organelle that you see. It looks like a bean shake, but it's actually a tubular network. These are tubes, and they respond dynamically inside the cell to both internal activities as well as external activities. You have to realize that at the time of conception, all of the mitochondria for the developing embryo are in the cytoplasm from the mother. -The cytoplasm. -The mitochondria are not in the nucleus. They're in the cytosol. -Oh, outside of the nucleus. -Outside of the nucleus, but in the cell, body itself. All of the mitochondria, they determine our destiny. They will determine how long you will live on the planet if you don't have an unfortunate accident or something like this. They have an expiration date. Different species die at different times. You don't find people living 400 years. It might live about two and a half years. Elephants live as long as we do or whatever. But that's all determined by this organelle. So you can see I have wrinkles and this kind of thing. This is from living on the planet. And this is from wear and tear on this organelle, which allows us to make energy efficiently. So when this organelle starts to falter with age, you die. You die from old age. This organelle has to be protected and respected if you would like to live a normal lifespan. But in diets and lifestyles and way we are today, we damage this organelle. And this organelle then can present itself damage to this organelle, which is a tubular network inside the cell. But let me say something else. It not only controls the internal environment of the cell, it also controls the neighboring cells, the liver neighborhood, the lung neighborhood, the colon neighborhood, the brain neighborhood, the glial neighborhood that went around the neighborhood. But they all come from the same origin in that cytoplasm. And they determine the overall metabolic health of your body. It's a systemic. They communicate with each other across cells, across tissues. I'll tell you, this organelle controls a lot of what goes, what that nucleus does. It tells the cell when to divide. It tells the cell when to slow down. It's kind of like a brain, but also like an engine room. It's kind of like that. Certainly, certainly the brain part of it is really mysterious in the sense of how it controls the destiny of the cell in the body. So sickness, sickness, disease, cancer, what do we know about the role that this little thing plays in these chronic diseases and illnesses and cancers that so many people suffer with? Yeah, well, this is the organelle that becomes damaged. And it can be damaged in many, many different ways. For cancer is what we have spent a lot of our time on. And now we've moved into the whole chronic disease issue, because each chronic disease can have different manifestations of ill health to the mitochondria and a particular population of cells. But in the case of cancer, which is what we call the most serious of the chronic diseases, creating the most trauma, the most emotional distress. But we have clearly shown, based on many, many works, that any multiple things from our environment can damage this organelle in a particular population of cells in a particular organ. For example, when you talk about carcinogens, it's a chemical that causes cancer. How does that chemical cause cancer? It damages the proteins and the lipids. These little squiggles are delicate internal membranes. They contain the proteins and the lipids that allows us to generate energy when we breathe. Okay, you're breathing, I'm breathing. I take in oxygen. Oxygen serves as a final acceptor for electrons that allows ATP to come out of this little, and don't forget it's a tubular network. And ATP is the energy currency. It's the chemical energy currency. It allows us to enzymes to work, allows all the metabolic machinery inside of a cell to work optimally. So just to play this back to you, so I know I'm clear. Oxygen comes in because I breathe in. I don't eat food. In that mitochondria, it does a process, and it spits out ATP as the energy. Well, and the waste products of a good energy metabolism would be CO2 and water. So when we burn gasoline in an engine of a car, we break down the octane, the carbon hydrogen bonds in an octane, and we have an internal explosion that drives pistons. Okay, the exhaust is a lot of waste products from breaking down. We're doing the same thing inside the cell. We're combusting carbon hydrogen bonds. And that combustion of a carbon hydrogen is a graded process. So it's not an immediate explosion. You're breaking down the carbon hydrogen bonds in a very precise way, producing ATP, which then drives the entire machinery of the neurons and the rest of the body. It can respond dramatically to energetic stress, emotional stress. Anyway, I gave you an example of a carcinogen, intermittent hypoxia, like people who have sleep apnea, they stop breathing for 30 seconds or more in that general. And then that creates Ross, ROS, and that's what carcinogens do, ROS. These are called reactive oxygen species. They damage those delicate membranes. And then what happens if it's too acute, too stressful, the whole cell will die. The cell loses its energy and we get apoptosis or necrosis cell death. But if it's gradual, chronic over years, months, years, this organelle loses its energy. its ability to produce sufficient energy, but the cell compensates interestingly enough by using these ancient pathways, heirlooms of our evolutionary past. So, because all life on the planet evolved without oxygen in the dark, these cells grew like crazy. There was single cells, there was unbridled proliferation and all this kind of stuff. They didn't have mitochondria. They had bacteria. And the bacteria, which this organelle came from, was a fusion between one cell that had a nucleus and was fermenting through the cytoplasm. And this bacteria, which is the mitochondria, came in. And now you have two different forms of energy. You have the energy in the cytoplasm, the ancient fermentation. And then you have this new form, which can take in oxygen and generate energy much, much more efficiently than the aerolumic. Well, listen, one of our big discoveries, if you can believe it. But you see that space in the middle there? That's called the matrix. And that's where the crebs cycle, the TCA cycle, which breaks down the food that we eat. But they have an ancient part of a fermentation mechanism inside, because before oxygen came, every all life forms were fermenters. Because there was no oxygen. We had to wait for those bacteria to make oxygen through a photosynthetic process. But all organisms were fermenters in the beginning. After this organelle came in and was able to take in oxygen, make energy really, really quite. But in that matrix, they have in the cycle, there's a little pathway there that makes energy without oxygen from the evolutionary past. So we have it in the cytoplasm of the cell, because they can ferment in the cytoplasm. But this organ, our big discovery with the work of Christo Schunoppel is from Semmelweis University, is the world leader on that little pathway. When this organelle becomes impaired, these ancient pathways of energy through fermentation arise. Okay? They try to replace the lost energy from the efficiency of this organelle. That space starts throwing out ATP from glutamine. It's another fermentation fuel. It's making ancient energy in the sophisticated organelle that evolved to make efficient energy. So let me tell you. So you asked me what about damaging this organelle and what happens? If the damage to oxidative phosphorylation, which means energy through oxygen, it's too acute, the cell will die. Cyanide is a perfect example of this. You take a mouse or a rat or a person and you drink Kool-Aid, the cyanide-laced Kool-Aid. You die because what happens is that cyanide binds to the protein that's going to use oxygen for energy and the whole system shuts down. What's the efficacy, basically? Yeah, you die instantly. And there are other things that can kill quickly, aside and a variety of the chemicals. But for chronic diseases, it's usually not an acute stress on this organelle. It's a chronic stress. And in cancer, what happens in a particular tissue, whether it's bone, lung, bladder, brain, they gradually compensate with these ancient fermentation pathways. So this thing, this organelle signals to the nucleus, I'm suffocating, I'm not getting enough energy. So the nucleus turns on the transporters on the surface of the cell to bring in fuels that will elevate energy through what we call oxygen independent mechanisms. This is an oxygen-dependent organelle. This gets most of its energy because we breed all of our cells are using oxygen. And the CO2 that we're blowing out and the water that will be collected in the form of urine when you put other waste products in there, that's efficient metabolic homeostasis. This organelle makes not only the cell, but the whole body in a state of metabolic homeostasis where all systems are working at optimal efficiency. But with chronic disruption, smoking, lack of exercise, you can go right down the list of all of the different things that can elicit cancer. Any kind of carcinogens, microplastics, forever chemicals, glyphosate, any of these kinds of things that would chronically damage the ability of this organelle to produce energy, viruses, oncogenic viruses, inflammation, you have chronic inflammation. Any of those things damage the sophisticated ability of this organelle to produce sufficient energy. That's why the oncogenic paradox, which we solved. What's the oncogenic paradox? That was the paradox. That was first put out by Albert St. Georgi, Hungarian scientists who received the Nobel Prize, I think, for vitamin C. He said he was very interested in cancer and living systems. He said there's a paradox. He said we know multiple things in the environment can elicit cancer. We've identified these oncogenic viruses, chronic inflammation, carcinogens, intermittent hypoxia, rare, rare germline mutations. He said we don't understand the common pathophysiological mechanism by which any of those provocative agents would elicit dysregulated cell growth, which is cancer. When you talk about cancer, what is cancer? It's cell division out of control. This dysregulated cell growth, this organelle determines when cells should divide and when they should not divide. It regulates the destiny of the cell. What happens when this organelle becomes chronically impaired? It falls back on these ancient pathways, these pathways that existed before oxygen came into the environment, where all the cells were dysregulated in the growth because they didn't have this regulatory system, which is the mitochondria coming from a bacteria. Let me play that back to you in a way, just to make sure I understand. I know it can be kind of deep. Okay, so in this mitochondria here that I have in front of me, there is an ancient, because this came from the fused. A fusion of bacteria is a long, long time ago. Yeah, that old bacteria. About several billion years ago. It used to be very selfish and just think about itself. It used to grow on its own and didn't communicate with anybody. Had its own way of growing and multiplying that was really not in cohesion with anything else. That is still in there somewhere. Although now in its modern form, because there's been that fusion, it grows thinking about the wider organism. When it becomes damaged, it falls back on that old selfish way of growing. That's kind of what can cause cancers. If there's stresses on this, which could be all the causes, cause and genetic things you described, then sometimes this falls back on that prehistoric, selfish way of growing, where it doesn't think about the wider organism. It's close. In the sense of thinking, we don't know about that. We just know the consequences of what happens when it falls back on those ancient pathways. Now here's the situation. People knew from what our auto-warburg said cancer is an energy problem in the cell. Why would the cells start to ferment and produce a massive amount of fermentation waste product, which is lactic acid, even in the presence of oxygen, 100 percent oxygen. They're still fermenting. That shouldn't happen. When you and I hold our breath or have a heart attack, let me tell you something. This is really remarkable. This is another piece of information that got us on this whole thing. When people have heart attacks, they stop breathing. The heart sees us. The bloodstream immediately fills with these fermentation waste products, which are lactic acid and the other one, which we now know is succinic acid. Okay, wow. These two waste products. Now if you don't start breathing in a short period of time, you're going to be dead. You die because the neurons in your brain cannot sustain this kind of fermentation energy for very long. As soon as you give them cardiac massage and whatever the guy's heart starts breathing again, the lactic waste products of lactic acid and succinic acid go away. They disappear because you're breathing now. You don't need to ferment when you have oxygen in the environment. The mitochondria can utilize the oxygen in the environment. Cancer cells, to Warburg, say, it's the weirdest thing. These cancer cells continue to ferment even in 100% of oxygen. Why are they doing that? And he speculated that this organelle was damaged, irreversible damage to the organelle happened in these cancer cells. He didn't have an electron microscope at the time. He didn't have the sophisticated tools that we have today. So he projected that all on biochemistry at that time. He was a biochemist and he said, you don't should not produce fermentation if you have oxygen. You should shut that off. And these guys should return to a normal metabolic homeostasis. And he said that's because there's something irreversibly damaged in this organelle. So a lot of people, attack them and they said, "Oh, we don't have any evidence for that." Here's the beautiful thing. Some cancer cells continue to take in oxygen and make ATP. Therefore, a warberg must be wrong. Uh-huh. We show that. We show that cancer cells take in oxygen, but it's not making energy through ATP in any great amount. It's using it for ROS, react, these radicals that further damage and cause the DNA mutations that everybody is chasing. It's all downstream effects of damage. So, what does this mean in simple terms? Because I'm aware a little about this. Well, it means I say, "Okay, so I have dysregulated cell growth. That's ultimately what the problem we're dealing with. We can't control how these cells are dividing with throwing all kinds of crazy stuff at them, trying to poison or radiate, surgically remove them. We're trying to do everything to stop this dysregulated cell growth." So when we look at this organelle under the electron microscope, we find these criss-tay are often missing. You have what they call ghost mitochondria. You've got the shell, but nothing inside. Or if they're inside, they're all deformed. So we know there's a foundational principle in biology. Structure determines function. If the structure is abnormal, the function will be abnormal. This is known to all biologists except oncologists. They don't seem to understand that. But it's an oncologist. Those are the people who study cancer. So what are you then? I'm a biologist. You're a oncologist. Yeah. You know that if the organelle is damaged, you're not going to be able to produce energy efficiently by oxidative phosphorylation. Okay? I think you're a bit further down the road, Thomas, than a lot of my viewers are. For me, hearing that there's this energy engine in my cells, and there's trillions of them, or billions of them, and they also communicate with each other. And when this becomes stressed or hurt or damaged because of lifestyle choices that I make, energy production and inefficiency will change. And that could cause this to die or malfunction in some way. That is for me. I go, I've got it. Okay. That's a major step forward. And now we build upon that. Yes. Now we build upon that. Okay? So here's the situation. We've discussed cancer cells, all of them that we have ever looked at have defects in the number, structure, and function of that organelle. Okay? I've looked at, I published that big paper where I spent over a year of my time going through the, while the early electron microscopy literature, Warburg didn't have that opportunity because that technology was not there for him. So he speculated that based on the biochemistry. But I went back and I looked at these electron micrographs of mitochondria in various cancers. And they're all damaged. There is few of them that risked, Christa, are gone. I worked with some of the best like, Aristomendium Marillo, who is a world leader in beautiful electron microscopy of cancer cells. And you can see, and all the damage under his magnificently beautiful. And why don't we have a couple of papers? But let me tell you something else, Stephen. In the cytoplasm, these organelles are also in contact with other cellular membranes, like the endoplasmic reticum. There's a lot of we call organelles inside a cell. You have the nucleus. You have the mitochondrion. You have lysosomes. There is intimate contacts between some of the other membranes. Mitochondral-associated membranes, what we see. And they're also abnormal when you look at them under the electron microscope. The ones that's talking too abnormal. Yeah. The ones that you can see. The mitochondrion are abnormal and the membranes that contact them are abnormal. And that intimate contact, I'll get into the calcium signaling a little bit later, which controls the destiny of the cell. Why the cell is growing out of control? Well, well, first of all, it's fermenting. That means it's getting energy from sources other than oxidative phosphorylation. You can take a cancer cell and treat it with cyanide or azide or in absinzoid. And it's still living. It's still growing because it's not using the oxygen path. They're not using the oxygen, falling back on oxygen independent mechanisms, which are called fermentation. So I understand that to be that there's a malfunction in a mitochondria, which means that it no longer uses its oxygen pathway in a cell. It is sufficiently as it should. There's always some residual level. And it finds another way to make the energy, which is how it survives. Yes. And then it stops communicating with the rest of the cell. Well, it actually communicates with the nucleus to open up the floodgates to bring in the fuels that drive this fermentation energy. It gets more and more greedy. It has to because you're taking an organelle that produces energy highly efficiently, like 34 to 36 ATP's with oxygen. With oxygen. And now you're trying to replace that with fuels that give you two moles of energy. So it's inefficient. It's very. And then you get two out of the, so you're getting four. So you have to take in, in order to make up the efficiency, you must have a tremendous logistic, you must have tremendous supply of the fuels that will give us energy, which are glucose, the sugar, and the amino acid glutamine. Okay. And the bodies are loaded with glutamine. That's the most abundant amino acid in our bloodstream. And evolution provided that for us. Because if we stop breathing, we use that fuel to keep the cells alive. We, our gut is controlled by glutamine. Our immune system uses glutamine. What does glutamine? It's an amino acid. Okay. Broke. We make from food. Yeah. Which we can make from food, true. Or they call essential and non-essential amino acids. Essentials are from that we must eat. We must have certain foods that provide these. And as a result, we must have certain foods that are in the bloodstream. And we must have certain foods that are in the bloodstream. Let me test you. When we fall, the mitochondria falls to that prehistoric pathway where it starts making energy in a really, really inefficient way without using oxygen in the same way. It starts relying now on glucose and glutamine to produce that energy, and it starts relying on glucose and glutamine to produce that energy. And it starts relying on glucose and glutamine to produce that energy. So you have to compensate because let me tell you, if you don't compensate your debt, the cell, I mean the cell dies. When we look at the populations around the world that have the most prevalence of cancer, it doesn't appear to be some of the countries like New Year, Gambia, Nepal consistently rank at the very bottom for cancer incidence. Conversely, high-income countries like Australia, New Zealand and the United States have the highest rates of cancer. Why is that? It's because of our technology, we are still paleolithic men, and our biology has allowed us to store energy efficiently because of times of famine. We are now in a new environment where we have massive amounts of highly processed carbohydrates. Inactivity, emotional stress, we have poor sleep habits. You pile all those together with the exposure to carcinogens and whatever, and you chronically damage this organelle. And in some organs, you can get breast cancer if it's a lung, if it's a. Whatever it is, that organelle becomes chronically damaged in some populations of cells in a particular organ, and you can elicit this regulated cell growth as the result of that. You know, what I find is in the paper that we have with the chart. If you can keep your mitochondria healthy, just don't forget paleolithic men, our ancestors from 500,000 years ago, or modern men, like you said, in these countries, living according to traditional ways with minimal interference from modern diet and lifestyle issues, have a lower amount of cancer in general. And this is what Albert Schweitzer found, the famous humanitarian physician. He was specifically looking for cancer in African tribes. And he said remarkably, it's extremely low. What are these guys doing where Western society has a lot of cancer, and these Africans have living, living according to the traditional ways. So they have a lot of exercise, they're eating all organic foods. They're not under the same kind of stress or exposure to chemicals that a modern society's have. And you find out you have very low cancer. Like for example, dogs are all evolved from the wolf. Wolves in the wild rarely have cancer. The domestic dog, cancer is the number one killer of the domestic dog. What is the dog doing that the wolf? The wolf is out running around eating natural foods. The dog is in an apartment somewhere, gets a dog walker once a day, right? And the next thing the dog is obese and full of cancer. So it all comes down to what you do to maintain the health and vitality of the mitochondria that reduce the risk. And that's what this chart I'll show you has. Reduce the risk of damaging this chronically. Okay. So that can explain in large part why modern societies are struggling with chronic diseases. Not only cancer, we have diet type 2 diabetes, we have obesity, we have blood pressure. We have a whole even neuropsychiatric problems. If you can protect and keep this organelle healthy, you reduce risk. Now people say, well, cancer has to be genetic because we have inherited genes that put us at higher risk, like the bracket one for breast cancer and the leaf from any for a variety of other cancers. Our paper in this pile done by Bob Kaplan, he went through and looked at all the genetic risk that which papers it's one of the ones published in oncology. But none of these mutations are 100% penetrant, meaning that they're secondary risk factors. A primary risk factor would be every time that mutation is there 100% of the people because I work in TASACs disease, I work in more and errors of metabolism. Those mutations are 100% responsible for that condition. There's no gene mutation that's 100% penetrant. You have that gene, you're going to 100% good cancer. Most of them are what they call incompletely penetrant. So what does that tell us about the nature of. Well, then we went back, what Bob did is he went back and he looked at what every one of those gene mutations in some way disturbs the efficiency of oxidative phosphorylation in that organelle. In the mitochondria? In the mitochondria. We have all the evidence, all the risk factor, all the genetic. Just the front page there. I was going to show you the hard data, but you got it. So anyway, all of them damaged this, the efficiency of energy through this organelle. So that's like carcinogens. That's like viral infections. The viruses that have a toma and papilloma, their products will go in here and damage it, or they will replicate inside this organelle, screwing up the efficiency, causing a compensatory fermentation, causing the dysregulated cell growth through abnormal calcium singling, causing cells to no longer be responsive to their neighbors. Is that clear? If I can make you understand this, we can make everybody understand this. Yeah, so the reason I spend a lot of time asking why and asking for clarification is because when I do this show, I then go out into the real world and I meet the people that listen. Yeah. And one of the groups of people that listens are young offenders. And when I went and visited them, I pointed out the episodes that I thought would help them. Yeah. And a young, one of the young offenders said to me, I can't listen to that episode because the words you used were too big. And I remember thinking to myself, okay, I get that all the time, but guess what? Now we have AI. And you can take this statement and put it into AI right on the, and it'll, it'll, it'll, it'll dumb it down for you. So, so that's a tool that we previously did not have. So, so I use that all the time. I said, when you hear me speak like this, don't think I'm married, don't think I'm just, these are the terms that we use when you're part of the academy. Yeah. Okay. But we can take those terms now and AI can do a wonderful job and synthesize it. Oh, yeah, I know what he's talking about now. But without that tool, then it becomes like you said, why I can't understand anything. I've got 60 seconds and I'm going to show you how much I can get done because of our sponsor called Whisperflow. And for those of you that don't know what it is, it's a business I invested in that turns your speech into text in any app or device. I'm going to post into our Slack channel which rewards whichever team member conducted the most experiments this week. Hi, everyone. Here is this week's experimenter of the week. Congratulations to the Diode of CEO trailer team, which is Aunt, Liv, Dom and Cam. You guys have one. Okay, now I'm going to open Gmail. So here is one of our founders on an email chain that I want to connect my team with. All I have to say is add my team's emails and Whisper will do exactly that. Now a quick message to Juan, who does my schedule every single week. Hey, Juan, could I record on Wednesday at 2 p. Actually, no, do not let's record at 3 p.m. on Wednesday. Whisperflow is four times faster than typing and it is incredibly easy to use. So if you want to give it a go, all you have to do is head to whisperflow.ai/diven to download it today. If you know me and if you've listened to this podcast before, you'll know how obsessed I am with sleep. I've been on a bit of a journey with sleep. I see it as the single most important thing that I have the privilege of having control over. And that's exactly what our sponsor 8 sleep are. It's a very, very intelligent piece of material to sleep on. Here's what the 8 sleep pod does. It monitors your body while you're sleeping. And with their AI, it adjusts the temperature for you using billions and billions and billions of hours of sleep data. And members have reported up to a 34% deeper sleep and falling asleep up to 44% faster. Me and my fiance, Melanie, sleep on different parts of the bed. And it adjusts my part of the bed to my body and her part of the bed to her body. So if you want to learn more about this pod, go to 8sleep.com/diven. Big opportunity. I'm not kidding. Give it a try. I really want to just make sure I take off the box of cause factors. So you mention stress. You mention sleep. I currently don't have cancer, it's rather than way I could for a bit. But I want to do everything I can to prevent the probability. And from what I've understood, that means protecting my mitochondrial function. By living a lifestyle where I'm not creating this sort of like crazy oxidative stress on the mitochondria. Right. So give me a prescription of how I should live my life to keep my mitochondria healthy. Well, that's why we developed the glucose keen to ketone index calculator, the first biomarker tool that can allow people to know the level of health in their mitochondria. Because when you shift from carbohydrate fuel to lipid fuel, what's lipid fuel? Fat. So ketones. Ketones are a water soluble breakdown product of fatty acids. Okay. So we store adipose tissue. We store fat, which is the belly fat. All over. We have a fat on the outside. Yeah. So on the outside, that was there because as a species, we had to survive in the most harsh environments. And food was not always there. So we had to survive all kinds of famine, all kinds of absence of food. Our bodies are so that I'm, it's a machine that was home over millions of years to be super efficient. So glucose is gold sugar. Okay. You either burn it or you can store it as fat. Okay. That's that's the key. But when you have, you're not bringing in sugar, that stored fat now moves into the bloodstream, goes to the liver and it's like taking a branch and putting it at a chopper and you outcome these little soluble ketone bodies. They're breakdown products of long chain fatty acids. They can replace sugar for the brain, for the muscles, for most other cells in the body, except the rithrocytes. But they can replace, they can replace the energy of glucose. Okay. So and that's how we evolved. But we're now in an environment where we have massive amounts of highly processed carbohydrates. And we, we don't want to pee them out unless you have diabetes or something. So we store them as fat. So we have an obesity epidemic as the result of our evolutionary ability to store energy, which kept us alive as this species because if we were unable to store the fat in 500,000, we would have all been extinct. You and I, this conversation would never exist. We would never have existed as a species except for our abilities to store energy. And we burn energy efficiently in these organelles. So this is all a very highly efficient machine. So when we lose that ability, when we have so much energy in the environment, stress and no exercise all this, we store more and more fat. We produce an environment that's very damaging to this organelles. But I can say, how does stress impact that stress elevates corticosteroids? When you're under stress, you get into a fight, into an argument or you're stressed out by business deal going bad or whatever. corticosteroids elevate elevate blood sugar, contributing to systemic inflammation. It's okay, for a short period of time to be pissed off at something. But it's the chronic stress. The chronic, like looking at the cell phone, doom scrolling, all this kind of crazy stuff. While you eat the big twinkie, doom scrolling twinkies, not moving. All of this creates stress on this organelle in some population of cells. It makes it work harder. It's damaging because you produce reactive species that damage the efficiency of this organelle to produce energy effectively. And that either will kill the cell, gradually, if it can't use compensatory fermentation or pre-discoge you to cancer. So either way, it's unhealthy. So we have chronic disease. Cancer is the number one big dog in the chronic disease world. I mean, it's the one that people fear the most. Type 2 diabetes, cardiovascular disease, dementia. All of these are part of damage to this organelle in one way or another. And like I say said, for Parkinson's disease, when that organelle gets damaged, the cells of the substantione, I agree, die. They are incapable of compensating with fermentation. So they up and die. Cancer is very rare in neurons of the brain. Neurons, they glial cells of the brain form these brain tumors mostly. But neurons can't compensate with fermentation. So they die. So you either get compensatory ancient fermentation leading to dysregulated cell growth, which we call cancer, or we get cell death leading to chronic diseases. What about sleep? And what's going on with sleep that's causing? Sleep is a way we can restore the energy efficiency of the mitochondria. If we're well slept. If we have good sleep. Yeah. You know, everybody feels good when you have a good night's sleep. Your body feels rejuvenated because you're not you're not stressing out. You're reducing the ability of this organelle to manage the metabolic environment. If you're up all night and you're stressed out, you're never given this organelle in a particular cell, in a particular part of the body. You get neuropsychiatric problems. You can get digestive problems. You can get cancer. You can get tytoid diabetes. You have a hole and we put it in the paper. They're all the different stress, all the different things that can chronically or acutely damage oxidative phosphorylation. So sleep basically gives the mitochondria a little bit of a break. Yes. It gives your whole body a break. Let's be honest. So, but you pile those things on together. Lack of exercise. I are ancestors. What do you think, our ancestors? How hard it is to run down and kill a big buffalo or a woolly mammoth? I mean, you're exhausted after doing something. As a matter of fact, you chase these animals. You separate, and this is another thing. It was really interesting. Come at an Israel, I think it was last year. They looked at the cavemen, what they were eating. They were eating the strongest members of the herd, leading to the indirect extinction of these animals. They found out that if you can eat the strongest member of the herd, you'll get the vitality of that or a buffalo or elephant or whatever the hell they were eating because they knew the marrow and the physiology of that organism at that point in its life could provide you with the strength that that had. Now, when you eat the strongest members of the herd because you want to be tough, you're putting the old and the young vulnerable to predators, leading to the extinction. And this is what a big paper came out of Israel. They looked at these cavemen, what they were eating, like 500,000 years ago, whatever they were doing. So, humans indirectly caused extinction of other species in part, in not all, in part because they were eating the toughest guys in the herd. So, because they felt that if I ate those guys, I'm going to be strong too. And in a way, they're right. But all of it has to do with the energy efficiency of your muscles, your brain, your ability to be resilient. In endurance, I'm telling you, these guys were chiseled. They weren't dying from type 2 diabetes, cancer, right? Dimension. They were dying from infections and injuries and child mortality, were mostly killing our paleolithic ancestors. But when you bring your body back into a low glucose ketone, you're actually going back, like you were, and this is why we developed the glucose ketone index. What is in that envelope in front of you? Well, this is a paper that's under embargo, because they think it's going to be, the world thinks it's going to be very important. And it is. So, and what it is, this is a way to keep that organelle healthy. So, this is a way to manage energy efficiency in the body. And this hasn't been released yet. It hasn't been released. Okay, it's coming out. It's going to be a lead article in the Frontiers in Science. And not only that, the paper, the paper was written for the scientists. And then the journal decided to make a second copy for, they call young minds. So, letting kids that are like eight to 12 or 14 years old, synthesize it down. And one of my colleagues said, that's probably what most people will be reading, because they don't want to know about the bioenergetics that actually goes on inside this organelle to explain why this chart means something. And you've been working on this for some time? Well, I built the GKI. So, let me tell you the story. There was a woman, an American woman, a lawyer, Trudy DuPont, who developed a kind of a brainstem tumor. And after I wrote my book, that book there, that's my only book. Anyway, Trudy wanted to use this metabolic therapy. She stayed alive much longer over 10 years with this. She eventually passed away, unfortunately. But so, I was measuring, because we knew that the tumor cells needed sugar to grow. Awe-Werberg showed that, many other people showed that. And they can't burn ketones, because the fatigued, you need a very efficient mitochondria to burn ketones for energy. Our normal cells can burn ketones for energy. And that gives us tremendous, we can actually breathe lower oxygen, more energy if you have efficient, if you can burn ketones efficiently in this organelle. But if the organelle is damaged, they can't use the ketones. They can't burn fatty acids or ketones, which stores lipid drops. It's one of these big papers here. These, Steven, you can't believe how people misinterpret information. They see droplets of fatty acids in the side of the planet. You see the cancer cell and these all that fatty acids? No, they can't. It's there to protect them. If they try to burn it, they blow this up and die. So there is a storage of fat. They can't burn fatty acids or ketone bodies. So I knew cancer needed glucose and I knew cancer couldn't burn fatty acids or ketones because this organelle is broken. So I'm measuring glucose and ketones independently in truity. She's doing the finger prick thing, sending me the information back and saying, "Oh, here's my glucose, here's my ketones." Okay, so I have a ketone, because we're in front of me. Right. If I put my blood on this strip, it tells me my glucose levels. If I put my blood on this strip, it tells me my ketone levels. Right. But if you look at them independently, glucose is very volatile, very variable. And this is why I developed a glucose ketone ratio. Because truity had a parking spot that was for handicapped because she had a cane. Her brainstem, Gleoma, was preventing her from walking as effectively as an organ. So somebody took her parking spot. She was bullshit. So she ran upstairs and took her blood sugar and it was 186 milligram per decaliter. It was very, very high. So I know she was on a ketotic diet and she emails me and says, "I'm going to die. My cancer is going to grow fast." What's going on? She says, "My blood sugar is like 186. And you know, it's supposed to be you told me it was supposed to be 60 or 50, 65 or that so." So I said, "What's your ketone level?" Oh, it's still, you just had.4 millimolar, or I think it was.9 millimolar. I said, "Well, that didn't change, right? No, just the sugar changed." So I said to the students that were working with Josh Feinberg, I said, "Josh, just trying to measure these two independently of us as a bullshit. You can't. This is hard to figure out." So what we decided to do in millimolar, because glucose comes out in milligram per decaliter, whereas ketones come out in millimolar. So we had to convert glucose to millimolar and divide it by the ketone in millimolar. And then you get a number that's not all over the world. It's very stable. So we were able to, because of true to that one cancer patient, we developed the ratio of this. Then later on, we realized that this ratio is a statement of how healthy your mitochondria actually are. So when you have these low ratio, you're paleolithic man. You're back in the zone where we didn't have chronic diseases, because we didn't have damage to the organelle that would cause those diseases. So paleolithic man, think, where are they getting their pastries? Where are they getting their cakes and sweets and all this other guy? They didn't have it. They weren't that because of choice, they were there because of circumstance. So, and we learned, Paleolithic man was always in some sort of a state of some kigtosis, because they wouldn't have food for periods. They were very active in their exercise. They didn't have chronic diseases, but they had other kinds of diseases. So, then my student, Derek Lee, myself and Christos Chernopoulos, we started to make a ratio chart. Now, these are the numbers that you get when you divide your sugar by your ketones. Okay, so my sugar by my ketones. So, if I did my glucose measure now on this, it'll really be. Okay, let's see what your GKI is. Oh, you want me to do it, okay. Okay, what you had a glucose, you had a ketone with 0.4 millimolar. Yeah. Okay, what was your sugar? How much? So, we can divide, we can do the division right now and tell you what you have. You can get these little ketones. Oh, yeah, it's a keto modra. And you can also get them now for, for, wow, this guy's skilled at doing this. I travel with one of these. Believe it or not? Yeah. So, I have one all the time. Well, 90. 90? Okay, so you have to divide, you have to divide, you have to divide. You're not getting. Doesn't this give you the push button and give it the GKI right away because the new ones have it. So you have to divide 90 by 18 and you get a number. Five. Five. So, divide five by 0.4. 12.5. Okay, so here you are. 12.5. You're down here in the, in the, in the prevention zone. Ah, nice. Okay, so, so you, this is where Paleolithic man mostly lived. Paleolithic man lived in the yellow green zones because they didn't have access to all of the things that would drive up your blue blood sugar and keep your teeth. When your blood sugar goes through the roof, your ketones are really low because insulin is now driving it up. So, so that's good. 12, huh? So I did the, I did the carnivore diet for a week eating big rib eyes. You like rib eyes steak? Yes, of course. Okay. Okay, rib eyes, bacon and eggs, lamb. I, so I did it for a week and I was able to get down to 10. Okay. And I couldn't get lower but I was loving the rib eyes so much. I ate too much of it, right? You have to be, you have some level of discipline. So, so, but people, people, this is what we call the zone of prevention. It's very hard to get cancer or chronic diseases when you're in these zones because you're keeping this organelle quite healthy. When you live in these zones consistently. You don't have to live consistently because humans, we, we involve as a scavenger species. We would engorge ourselves because we knew it wasn't happening every day. Modern man is living in the feast every single day. Yeah. And that's why we're, we're have an up, all the chronic diseases. This is the red zone is the zone of risk for chronic diseases and cancer. And when you look at the obesity epidemic, you look at all these things, these guys are, oh, and it's, it's like we can visit the red zone. We don't want to live in the red zone. So if I was to visit the red zone, it would look like meeting high carbohydrate diets, lots of sugar. Yeah. No exercise. No exercise. Yeah. And basically modern man. It's like, I'm getting five meals a day, like snacking all the time. Oh, then you'll be, you know, we have, there's document cases, we have them up to 500. You can get your GK. GKIs of 500. You have people with, you know, blood sugars about four or 500 milligram per deck of liter. I mean, you could do the zero ketones. I mean, you do that. You do the math. It's, it's unbelievable. You're basically saying, I want to keep my blood glucose levels. And you want to, you want to have some level of ketones and high in my ketone levels, somewhat as high as I can. Well, it's, you don't want to go because people don't have the physicians listening to go, oh, he's going to go into ketoacidosis. You know, people, give me a break. Ketoacidosis is like when you have ketone levels of 15 to 20 millimolar. Are you kidding me? What is your zero point four? That's called nutritional ketosis. That's how we evolved. When you have type one diabetes where you can't control sugar or, or, or, or insulin, you have no insulin in responsive, you're going to get high levels of sugar and ketones. But this is a pathological condition. Most of type two diabetes, these are all pathologies based on damaging oxidative phosphorylation. So what this chart does is for the first time, and we put it together because we did all the, in the paper, it discusses the bioenergetics. What we're finding in cancer is that if you can get into the green zones where your blood sugar is low and your ketones are elevated, you hammer the hell out of these tumor cells because they, they, they, they, they're, you're taking away one of their two primary fuels driving dysregulated cell growth, okay? And as the, and as the, and as the ketones go up, the rest of your cells in the body are getting super healthy. The tumor cells can't tap into the value of a ketone because the organelle needed in the tumor cell to do that is corrupted structurally and functionally. Is, am I clear about that? Yes. Okay. So ketones will make you healthy, the normal cells of your body, but cannot be used to help the cancer cell because you need a good structural functional organelle to burn them. So they become marginalized. And if the ketones go up, they're actually toxic to that, to that cell, to some extent. So, but they're still alive. The cancer cells are now incapacitated. Where we, we showed you get rid of the abnormal inflammation, you get rid of the angiogen, the abnormal blood vessels. You're taking an angry tumor and making it much less angry, much less inflamed, more indolent, kind of a tumor, but it's still there. It's not because the other fuel that's keeping this cancer cell going is the glutamine, okay? So now when you have the patient in this green zone, this is for management now, Stephen, this is prevention is, is never having to deal with what I'm talking about. If you're living in the yellow zone, the probability of getting cancer or chronic diseases is already reduced, okay? But now you have some poor guy out there who's living in the red zone his whole life. He wants to manage the cancer that he has. He has to get down in the green zone and try to stay there as long as he can. But the cancer will still grow because it has access to glutamine, okay? And glutamine is always, here's the bloodstream. You have this much blood. Cancer needs that. So you always have this surfeit of glutamine. So you have to come in now with drugs. And the drugs like repurposed drugs to target the glutaminolysis. With this one here, this BMC, big paper here, okay? This paper and the new one we have with the ketogenic for the childhood, high grade gliomophic kids. Once you get down here, you come in with drugs that target glutamine. I've looked at one and that's embendazole, okay? How did I come to that realization? People knew that embendazole had some therapeutic benefit about cancer, but they don't believe until you show the mechanism. That paper shows the mechanism. The target's glucose and glutamine. That was this one. Target's glucose and glutamine. The two fuels driving the dysregulated growth of the tumor. Okay. So here's the mitochondria. So it's getting the glucose in the cytoplasm from the sugar and making and fermenting that. And then also the amino acid glutamine comes in. You have to block the glycolysis in the glucose, these two pathways. You have to restrict the availability of glucose and glutamine together at the same time. I speak only about things that I have tested in my lab and published papers on like this. So you have to realize that the cancer field doesn't understand that the cancer can't grow without glucose and glutamine and can't switch to fatty acids or ketone bodies. That's still not going to kill the cancer though, is it? It's just going to. Yeah, we don't ever use the term cure because some of these tumors. Now let me tell you, people like Pablo Kelly from Devon England, he had the glioblastoma. He didn't take any radiation or chemo. He just did metabolic therapy. He lived for 10 years. He was diagnosed with an inoperable glioblastoma. They wanted to irradiate and poison him with the drugs. He said no, he was one of these naturalistic kind of guys. And he lived for 10 years and the tumor became operable. He had four debulking surgeries on an originally described inoperable cancer cut out four times because once we put the metabolic therapy, the demarcation of the tumor, whoa, are there a surges? I think I can get this out. But he never got rid. he lived with it for 10 years, had a couple of kids. He died from a cerebral hemorrhage on the last debulking surgery. He never died from the tumor. But you're saying that the two work together in tandem. Yes. You're saying that the chemotherapy works in. Now that's another thing. So what we do. That is very interesting. So if you put the patient in nutritional ketosis, the ketogenic state of nutritional ketosis facilitates the delivery of drugs to the tumor cell. It actually makes. you can use lower doses of drugs and you get bigger effect. The therapeutic benefit increases with lower dosing. So you want to be in ketosis when you do these chemotherapy. Yes. and then you use much lower doses. This is what we're doing in a stimbul clinic. We're taking pancreatic cancers. These guys live in four and five years. What are we doing in advanced breast cancer and all these terminal cancers? We put them into some level of ketosis and then you come in with the standard drugs, cisplatin, carbol plat, whatever you want to do. But you're. the doses just down big time and then they have tremendous and you put the key, the, the, the, this is what the title of the paper is. Keterogenic diet as a metabolic vehicle for enhancing therapeutic efficacy. The current body of research suggests that being in a state of ketosis can act as a helper therapy, enhancing the cancer killing effects of chemotherapy while simultaneously protecting healthy cells. Yeah. Yeah. Right. And so that's what we're doing in the end of the day. So that's what we're doing in the end of the day. Immunotherapies, you hear about these things, a chimeric acid and T receptor, PDL, PD1, PDL1 and Ivers. They're called precision medicines, right? So look, they're designed to attack a molecule on the surface or stop that cell from being resistant. And if you were trying to attack, and what they do oftentimes, they come at, after you failed chemo and radiation, they then come at you with an immunotherapy. The metabolic pressure shrinks down your tumor, makes it very indolent, non-aggressive. And the rest of your body is healthy. And not going bald, and not bleeding gums, your microbiome isn't blowing it to hell. So, and then you can come in with low-dose chemo, immunotherapy, because whatever left in that remaining residual mass, they may all have something in common for having survived all this, right? So now you can come in with a precision medicine and possibly resolution. I thought this was fascinating. It says chemotherapy creates massive oxidative stress by damage inside the tumor. To repair the damage and survive, the cancer cell requires massive amounts of glucose. So if the patient is in ketosis, the tumours glucose supply is essentially cut off. The cancer cell can't repair the DNA damage caused by your chemo, leading to faster tumor death. Yeah, interesting. Well, don't forget, also, listen to this. And as another thing, people go in. What protects the tumor cell from chemo and radiation is the waste products of fermentation, the lactic acid and the succinic acid that are dumped out of this raging beast, prevent these other therapies from working. So if you want your therapy to work, you got to target those two fuels together at the same time. And when you do that, now the shell does off. These things are super vulnerable to even load doses of chemo and radiation. And the immunotherapies, look, if they have an immunotherapy, you try to attack the beast when it's at its strongest. You're not going to win. And this is what happens. You get only partial response. They, in the field of cancer today, they think living an extra six minds is a major breakthrough. We're talking about living an extra five and six years. This is what's really important. I just, I was reading some research to figure out if oncologists, so cancer doctors, are currently recommending the ketogenic diet. And it says, the vast majority of mainstream oncologists do not recommend the ketogenic diet to their newly diagnosed patients. In fact, if a patient brings it up, many doctors will actively advise against it. And the reasons for that, number one, is the fear of ketxia? Ketxia. Ketxia. Cancer Ketxia is a severe wasting syndrome where patients rapidly lose muscle and fat. It is a massive problem and a leading cause of mortality in cancer patients because the ketogenic diet suppresses appetite. And often leads to weight loss and colleges are terrified that a strict key to diet will accelerate Ketxia and weaken the patient. Well, that's because they, they have not heard what I just said with respect to the biology and biochemistry. Ketxia, there's two ways you can lose in cancer patients. Ketxia is the ability of the tumor cell to mobilize energy out of the muscles. It's taken the glutamine out of your muscles and feeding. This is one of the two fuels that's driving the beast, is glutamine. Where they get the glutamine from, they're getting the glutamine not only from the bloodstream, but they dissolve your muscles as part of that, part of that process. So when you put a patient in nutritional ketosis, the weight loss is therapeutic weight loss. Ketxia is pathological weight loss. So either way you can lose weight, you should take a high dose of chemo therapy. I travel all the time. I made a rule in my life that I'll only travel with a cabin bag. The problem with this is not much space. And here's the solution. It is called an extra 1% travel pack. And I teamed up with extra to make this. Typically I can only bring some of my black shirts. And it's a trade off of which black shirt should I bring. How many of these do you think I can get in here? So let's try 1, 2, 3, 4, 5, 6, 7, 8, 9, 10, 11, 12, 13, 14. Let's try 15, 16, 17, 18, 19, 20, 21, 22, 23, 24. Okay, so that's 24 black t-shirts. Here's where the magic comes in. Zip it up. Make sure it's nice and sealed. And then use this little contraption. Stick on there. I can now take more than 20 black shirts with me, which will last me three weeks. So if you travel frequently and you want to get one of these, you can save more space and be away for longer. Go to extra.com and use code DOAC for 10% off our collection. And I think some of the background context here is that mainstream ecology operates on the sort of somatic mutation therapy, which is the belief that cancer is fundamentally a genetic disease driven by DNA mutations. And because they're training focuses on genetics, their treatments are designed to target DNA and cell division, like human radiation targeted genetic therapies, rather than manipulating cell cellular metabolism. And lastly, as mentioned, I think we talked about this earlier, mainstream medicine requires massive multi-centred double blind phase three clinical trials, but for a protocol becomes the standard of care. So dietary interventions rarely get the level of funding. So oncologists lack the institutional green light to prescribe them to patients. And they tend to say to them, eat what you can. I don't blame them. There are good, many of them are good people. The problem is the system doesn't train them to understand the biology and biochemistry of the disease they're treating. Some of them become very resistant to say, say, get angry because why didn't I why wasn't I told this? Well, first of all, they're not reading these papers. You asked them about it. I never read it. Well, how are you going to know anything if you don't read the literature? Listen to this. The national cancer Institute, the NCI on their website on their website says cancer is a genetic disease caused by what you just said. Okay, they what I said, why don't they put the articles in there showing all the tumors that can't find any mutations. The somatic mutation theory says that cancer is caused by random genetic mutations. So now, new sequencing of normal people like you and me are finding mutations in all these driver genes and cells in our body that aren't disregulated cell growth. So we're calling them wild type cancers. We mean a wild type cancer. You lost me. Okay, the nucleus of a tumor cell, a raging tumor cell, which causing him that cell to grow is that the mitochondria in the cytoplasm or is it the mutations in the nucleus? Okay. So the according to the somatic mutation theory, it's the mutations in the nucleus that are causing the disregulated cell growth. It take that nucleus and put it into into an enucleated normal cell. Yeah, and there's no cancer. No, can't there's no dysregulated must be something else. Then you take the nucleus of the normal cell and put it into the cytoplasm of a tumor cell. You get dysregulated cell growth. So it must be something other than the nucleus. Yes, the mitochondria. Interesting. It says so with mitochondria controlling our destiny and and the field of cancer has yet to understand it, accept it. And then say, well, we can't do any of this until we double-line cross. So that's just what do you mean? The science is telling us this. That's your way of protecting a broken system. Are you pissed off about this because you do seem pissed off about this. Well, who wouldn't be? Steven, there's 1,700 people a day in this country dying from cancer. I don't know the English. Listen, that comes out to 70 an hour. And it gets worse every single year. And the last time I was on this show, you guys use some old data. Look it up today. It's 2026 American Cancer Society says this year in 2026, we will have 626,000 souls leave the planet from cancer. And every year it gets worse. So when you hear all the breakthroughs, we have, we have television ads in Boston for the cancer system. Breakthrough after breakthrough after breakthrough. All these different, they come on. And all we do is get more dead cancer patients. Raise money for cancer. Where's the, where's the accountability for all the money you're raising? When are the work? When is the people going to wake up? You don't make someone healthy by irradiating and poisoning them. You've got to understand the biology and the biochemistry of the disease. I have the concepts and the proofs. But the physician who works with the patient on a clinic basis, they're the ones that must apply this to the clinic. So there's two different things here. is the hard science that That's the bedrock for this. And then there's the clinical person who has to practice that practice. That's that. Together you get great success or better. I say success better than anything that's out there today. The American Cancer Society recently released its latest projections for 25 and 26 and the data paints are fascinating dual-sided picture. More people are getting diagnosed with cancer. New cases the ACS projects over 2.11 million new cancer diagnoses in 2026. This translates roughly to 5,800 new cases every single day. Approximately 626,000 Americans are expected to die from cancer in 2026. Right. About 1,700 deaths per day. Lung cancer remains the leading cause of cancer death projected to cause more fatalities than colorectal and pancreatic cancers combined. I also just wanted to pick a whole point we were talking about earlier which is about the metabolic approach to cancers. It says here and this is going to the point about people telling you to eat whatever you want while you're managing cancer. Because the primary goal of the hospital dietician is to prevent weight loss during brutal chemotherapy regimes, patients are frequently told to eat whatever they can and eat whatever they can keep down. It is incredibly common for cancer patients to be handed meal replacement shakes which are often packed with corn serps and refined sugars, ice cream and high carbohydrate comfort foods just to keep their calorific intake up from a metabolic perspective. This is a tragedy while it keeps weight on the patient. It simultaneously floods the bloodstream with glucose insulin directly feeding the tumor. And while keto is not the standard of care, the landscape is beginning to slowly shift. There is a growing minority of integrative oncologists and specialised metabolic clinicians worldwide that actively prescribe therapeutic ketosis alongside conventional treatments. These doctors use the keto diet and fasting protocols to protect healthy cells and sanitize tumors before administering lower, more targeted doses of chemo. That's what we developed. That's our plan. That's what we're doing. Okay. We do that because we understand the biology and biochemistry. And that's why we developed, we're developing the new society called more, the more alliance, metabolic oncology, research and education. This is bringing together what you just mentioned there in a logical approach to manage cancer. This is a logical approach based on the hard science of decades of research initiated originally by Otto Werberg and then continued by our group at Boston College. The American Cancer Society says that breast cancer is increasing, prostate cancer is increasing, pancreatic cancer, melanoma, HPV associates, oral cancers are steadily increasing in the incidents of severe cancers. Yeah. There has been no major advance in managing glioblastoma in 100 years. What's glioblastoma? That's the deadly brain cancer. Okay. That killed Teddy Kennedy from Massachusetts, Senator Kennedy, John McCain, President Biden, son, Bob Biden. It's killed a lot of various people and it's considered a death sentence. I don't know. No, no, no. We're keeping these guys alive. Okay. We're not saying we cure the cancer, but we can certainly keep them alive a lot longer. We're getting very, very excellent results in managing pancreatic cancer using metabolic therapies. We know what to do and we know how to do it. I have clinicians that work with me, dieticians that know how to manage cancer effectively. We can do it right now today. If someone were to say, "Seafried, get your group together. Let me see what you can do." I will put up our metabolic therapy against any trial from any of these pharmaceutical companies. We can keep these people alive a hell of a lot of people alive. We can keep these people alive a hell of a lot longer to participate in our society. We're not doing that. If you were made president today of the United States, of the United States, no go there. I'm going to go there. Those guys are not, let's put it this way. They're not scientifically literary. But you would have to be, yeah, right. Professor Thomas E. V. U.N. president of the United States, your primary objective is to bring down this 1,700 Americans that are going to get cancer a day. Is it get cancer or die from cancer? No, they die from cancer. 1700 a day dying from cancer in the United. 70 an hour. Think about it. You can put in place policies to stop this happening and to also help people manage it. But what is it you do? First of all, we wouldn't throw out everything. Just like I said, we have a strategy now to manage cancer effectively. Okay. You were so productive. You're going to get rid of the drugs that are making billions and billions of dollars. We're just going to use them at lower doses. Is it a different, different, different. But you also want to prevent it in the first place. Well, preventing it. I'm going to write it. That comes back to our chart. I'm going to write down the manifesto. So what is, what do we do to prevent these 71 people an hour dying of cancer? Well, that has to come from government policies. Okay. You just got promotion. You're now the king of the United States. So you didn't even need to ask anybody what you do to prevent the 71 people a day dying of cancer. It's going to be education. Education number one. Okay. You have to let people know that those. And now let me tell you another thing. It's really important. It should not be any government or government official telling anyone what they should or should not eat. Okay. The power of this chart is personal. You're emboldening the patient. They have to know. We're not going to tell somebody, oh, if you continue to eat bad food, you're going to have high risk. And that person. I don't care. I'll smoke cigarettes. Well, the government is not going to come in to this guy's house and take away the bad food. No, no, no. That should never happen. Those foods are there because they give us pleasure. But they should be the knowledgeable person would be to say, I like to have it every now and then, but I can't live in that environment. And the other thing we do terribly in this country, the poor people in these food deserts, where you only get crap food. And as a tragedy in itself, they go to whole foods where they have the expensive rib eyes and all this stuff. That's much more expensive. A lot of people can't afford the kinds of foods that will put them in these better healthy zones. So we're going to make food, healthy food more cost effective cheaper. Those are easy words to say, but in practicality, it's not. And what kinds of foods should people be eating? Well, I think they should just try to avoid the highly processed carbs. Okay, listen to this. An exercise. There's a lot of things we can do that would mitigate the inflammatory conditions put on this chart. The goal here is we know what keeps us healthy. It's the efficiency of that organelle. We want to do everything possible to keep that organelle healthy. We will reduce dementia. We will reduce diabetes. We will reduce obesity. And they say, well, GLP, why don't I want to do it? And human beings are the kind that I want to quick fix for everything, right? A Zempec. How about some Zempec? Is this what is this GLP? Yeah, GLP one. Okay, we don't, you know, first of all, we haven't done any research yet to know where a GLP would put you on the chart. We do know one thing at lower's blood sugar. How high of a level would it bring to ketone? Because it's the ketones that keep the organelle healthy. So I'm lowering blood sugar, but am I raising the ketones that enhance the bioenergetic efficiency of the organelle? I don't know what number has them done yet. I haven't seen any papers coming out. Okay, so we'll keep Zempec off the table, but you're saying exercise number three. So I've got education, kill the food deserts so people can get healthy food, stay away from the old process stuff. Number three, exercise. We're going to give everybody free gym memberships and whatever else they need. Reduce stress, emotional stress. You got to do that. There's a lot of ways, music therapy. There's a lot of different ways you can get these things. Meditation, friends, happiness, all of those reduced stress. So I had a guy from Korea. I think it was Japan or Korea. We had a big meeting one time. And he didn't tell me what to do. He says, "You want to get cancer? If your job and if your goal in life is to get cancer, you got to eat crap food all the time. You have to have terrible sleep. Make sure you never unask the couch. Sit in front of the TV all day. Make sure you never exercise and make sure you don't have any friends or be happy." He says, "You're on the fast track for not only cancer, but all these other chronic diseases." So that's what I just said. You need to not do that. You're doing a great job. You know, there's not many people that are going to be measuring their GKI every day. But there are people who love to measure that kind of stuff. I would say I have another thing. So they put these things on your arm. These continuous sugar monitors. They're making continuous glucose ketone monitors. So apps are coming out now. Believe me, there's apps I've, Lucas Liu and some others are making these apps in my lab. You can take your cell phone and photograph a particular food item. And the food item, immediately you put the places where the food item on the chart. So you'll know, "Well, give me what zone you'll be in if you eat it." So it's really interesting. And these things are coming. We're using AI to. But it's purely patient empowerment. The patient themselves, the person themselves, make the choices. No government, president or king or whatever you want to say, should ever tell people what and how they should eat. The patients should be familiar with this and have the knowledge to know. I'm going to test what I think. So people say to me all the time, Just tell us what you can eat. That's all they say to me. Okay, eat whatever you want. You figure out where on the chart you're gonna be, and then you'll know. So people say, "Well, I can't eat ketogenic diet." Well, our group in Greece, now you tell me, they got the brain cancer tremendous success in keeping brain glioblastoma guys alive. What was the diet? It was that calorie-restricted Mediterranean diet. Salmon, sardines, olive oil, avocado, and exercise. Is that like, oh man, that's the worst diet. What about the carnivore? What about the ribeye with a little sauce per naeze on top of it? This keeps your blood sugar low and elevates your ketone. If you want to do it with plants, everything, you got fish, plants, you get the vegans and all these kind of people. This is a bio-energetic roadmap to health. Let me just give some specific sayings. This is amazing. What about high fructose corn serps and refined corn? Oh man, that's the worst kind of crap. You don't take that. What about industrial seed oils? Well, people talk about seed oils. Canola and soy. Yeah, I don't know. And listen to another thing too. You and I are different. We have an individual metabolism, age, race, sex, all the religion, all kinds of stuff, determine what and how you live. And I can't be sure what you eat or what you exercise where it's gonna put us on the chart. Sympatic pesticides. I was really here that it increases the chance of lymphoma by staggering 41%. They all damage the oxidative phosphorylation, putting the cell at risk for compensatory fermentation, dysregulated cell growth. Wow. The problem, the problem that field doesn't understand what I'm saying with respect to the origin of cancer, how it happens mechanistically, how this organelle controls the life of the cell. They don't know enough about the biology and biochemistry of the mitochondria. You wanna get guys on here, like Nick Lane from England. I mean, these guys like Doug Wallace and some of these guys, they're mitochondrial biologists. They understand this kind of stuff. I wanna give people actual things that they can think about. So that's why I was asking you this question about you becoming king, fasting protocols. Well, okay. Intimidate fasting. Let me talk about that just briefly. Do you ever try it? Yeah. What do you think? You like it? It depends how long you're talking about. Okay, let's go a week. Uh, no, I'm not fasting for a week before. Okay. You know what? It's the call of the wall. This guy, he just sent me his book. It's coming out. Very nice guy. A viral Semic. He say people share things with me. The wall is after about three days of not eating just drinking water. You hit this wall and it's like, oh man, I'm just, I can't deal with it anymore. It's just a terrible feeling in my body. I can't sleep at night. I got the Jimmy legs. I got all kinds of problems. Screw it. I'm not doing this. I'm just going to get out of here. Just tiny amounts of a grape juice you can get through the wall. What we do for the cancer patients in the way we design our clinical procedures, which my clinical friends, we do a zero carb diet for about a week while the body is readjusting, getting, getting, bringing them out of the red zone, getting into the yellow zone. You can't believe the power of glucose is an addictive drug on the brain. It's unbelievable. It's like cocaine. It's, it's, and you know that when you start, you start shaking and you go on, so, but if you don't eat carbs and just eat meat or whatever to keep you in a low G.K.I., then when you jump off to the water only fasting, it's much less traumatic to the brain. You've gone through the wall of the gate, so to speak. So once you know how to get through the gate, you make this whole process a lot, a lot easier. And that helps people enormously, especially those people that want to get rid of their chronic disease, okay? Because, or some, you know, we have a lot of people out there that just like to do all this stuff, you know, you ever get these, these people that go overboard on everything. But you know, right now we have an obesity, chronic epitome, cancer epidemic, all of these epidemics are the result of an abuse of that organelle in one way or another. And we have a plan to mitigate that abuse. And at least people haven't, at least they're empowered to do it with the help of knowledgeable physicians. I want to just keep on this point of actionable feedback. So Dr. Walter Lungoz, Dr., his extensive research clinical trials proved that fasting mimicking diets drastically lower IGF1, which triggers cellular autophagy and actually makes standard cancer therapies up to three times more effective by removing the metabolic shield of cancer cells. Yeah, which is the glucose, the waste products of glucose, which is the lactoc acid and the succ acid that all goes down. Would you recommend, again, if you're king, would you recommend that? If I would never recommend any, I have to just give them the knowledge, then the person has to make the decision themselves. Do you think it could be beneficial? Of course. If you were king too, have everybody wear a CGM. One of those continuous glucose monoces is at least once. No. Never. No, I think if you have cancer and you really want to stay in this green zone to know, right? Listen, I was down in Mexico and outlaw. I was talking to one of the head physicians down there. He wore one of these things, right? He said, every time I wanted to go out and have a party and have a good time, this damn thing would be beeping. So what do you rip it off and through it in the trash? It does. You don't want somebody barking in your ear when you're having a good time. That's why what you're doing right now with this, you're choosing to prick your finger. You're the one making that decision. There's not something on your arm saying, "Steve, I don't do that. I don't do that." But you know what? It was useful to wear it once or twice because it even, I suddenly realized that things I put in my mouth had an impact on my blood sugar and my blood, which was, also, I realized that it had an impact in 10 minutes. My blood sugar came back down and crashed. I thought, "Oh gosh, I feel I could suddenly pair my behavior to my feelings." Yes. Well, that, no, I'm talking about it. I know. I've seen that. Not only that, Andrew Scarborough from England, who's been doing this for either stage 3, Gleoma. He's like 15 years out. He's done this so many times with the finger prick and all this. He knows that already when his body is in these zones from the feeling that you just described. But for the people at the beginning and who are given a terminal diagnosis, they want to get into these green zones and they want to use the things that are going to keep them alive on the planet for a longer period of time with a higher quality of life. That is important. That thing on your arm can help you stay in that zone until you have this thing managed at which time you can choose when to do that. So this flexibility in this whole process is not one, like for example, we have the standard of care which is written in granite for crying out loud. They get all, if you do anything different from the standard of care, you could lose your license as a physician. Metabolic therapy is patient driven. It's driven on the patient. What about hyperbaric oxygen? Yes. There was a study in 2013 that demonstrated that while a ketogenic diet alone significantly slowed tumor growth in systemic metastastic cancer mouse models, combining the diet with hyperbaric oxygen therapy, elistidoprofans, synergetic, decrease in tumor growth and drastically increase survival times. Yeah. We published that paper with Dominic Diagostino. We put my. Listen, hyperbaric oxygen will create oxidative stress in cells that do not have efficient oxidative phosphorylation. Ah, cancer cells. Cancer cells. So you can kill cancer cells by oxidative stress by irradiating or poisoning them, or you can put a patient in nutritional ketosis and then put them in hyperbaric oxygen. And the cancer cells are selectively killed. When you irradiate somebody, you're damaging the whole body with all kinds of stuff. And there's another thing. Steven, you got to listen to this and you listen carefully. When you go to these treat these standard of care, standard. When you standard of care, radiation, chemo, whatever they get in, immunotherapies or whatever. So the patient comes in and says, "I've been really working hard on this." The doctor says, "Oh no, that doesn't work, right? Give you some radiation or give you. You're flying up into the red zone." The treatment itself puts some stress on the body. The body itself starts going. You go into the red zone from the very treatments that you're giving to the patient, which is making strengthening the tumor cells. You know, again, chemotherapy, though, aren't you? No. I'm positive about it, but it has to be used in the right context. It has to be used when your body is in this state of nutrition. You can use low doses so you don't force the tumor cell to become even more resistant to the treatment. The next thing, which is actionable, is what you talked about earlier, which is these microplastics and forever chemicals. In late 2023, the International Agency of Research on Cancer officially upgraded these forever chemicals, which are used in non-stick pans and food packaging, to a grade one carcinogen in humans, cancer causing inhumans, based on strong mechanistic evidence that it induces epigenetic, which is gene alterations and suppresses the immune system. So you'd ban the forever chemicals. Okay. This is beautiful paper. I went back and I took what all of Warrbring meticulously went through all of his work, showed where he was absolutely correct and where he just didn't have the new information that would make him. I talked about the forever chemicals, microplastics. Guess what? They damaged the organelle. They get into the. They break, they cause rost and damage, it reduced the efficiency of oxidative phosphorylation, causing a compensatory increase in the utilization of glucose and glutamine and dysregulated cell growth. Everything comes back to this organelle. All chronic diseases and cancer are the result of damage to this organelle. That's why having this little thing here is so important. It's a great prop. I ought to get one from my class. You can keep it. The next thing is purifying the water supply. Heavy metals are found in local supplies to run off. ultimately are cost andogenic in some cases. The IARC classifies arsenic and cacadium as group on costogenics. And they frequently found in unfiltered public water infrastructure. So you clean out the water supply as well. - Yeah. You know, all this stuff is coming into our water supplies, people flushing down all these chemicals into the, which then leech back into the water supply. And every one of the chemicals that we have looked at that has been linked to oncology or dysregulated cell growth all damage the oxidative phosphorylation chronically. So we're bringing the entire focus back to things. What can I do to keep this organelle healthy? Even if I'm exposed to these chemicals, if I can get into these zones like you're trying to do. So even if you are exposed to these, this organelle has an incredible healing power in itself. - What is the most important thing we haven't talked about that we should have talked about, Professor Thomas? - Well, I mean, you've covered a lot. One of the things I want to talk about is metastasis. - What's that? - And that's the spread of the tumor throughout the body. Okay, so if you were to have a cancer that's just localized in one spot, you know, the probability of developing a therapy that would be long term is highly increased. The problem that kills people is the spread. So if the tumor is in the breast and it spreads to the liver and the lungs and the brain, you know, you've got a problem. Long cancer spreads to the brain, the liver. Most of these cancers that spread to the brain or other organs become difficult and that's why you use systemic chemotherapy. You're trying to stop. What we have found is that you have a stem cell. People love stem cells. If you ever hear the term stem cell tumor, wow, stem cell tumor. Stem cell tumors cannot metastasis. How do I know? Because I have stem cell tumors, diagnosed as stem cell tumors with stem cell markers. I've grown them. They grow very angry. They get a lot of blood vessels, but they can't spread. Okay, how do you get spreading tumor cells in your body? The immune system comes in, recognizes that as an unhealed wound and then fuses with the stem cells and then you have these hybrid cells. They are programmed to move around your body. So they are a macrophage tumor cell hybrid. So, and they're very hard to kill. But we found they're remarkably sensitive. They're glutamine driven. So we know they're glutamine driven and they need the glucose. And that's why metabolic therapy done the right way can nail those metastatic cancer cells, purging them with a little bit of immunotherapy to go along with it. You might be able to get what we call resolution. Don't forget, my colleagues and I Dom Diagosteo, Joe Maroon, we built the press pulse therapeutic strategy. I mentioned that on your previous show. That's the way you press down the glucose of the tumor and then you pulse to kill the glutamine, which will target the metastatic cancer cells, enhancing the health and vitality of the organs already infiltrated by the tumor. I looked to an our previous conversation, Professor Thomas, and it's quite heartbreaking because the comments sections are all people that are either struggling themselves with cancer or are loved one of theirs. Their wife, their husband, has just been diagnosed with cancer. Is there anything for those people that have clicked on this video? Because I imagine they are in the millions. Yeah, they're living with them. They don't want them to hear. Well, the thing of it is is why, well, if this is a bigger issue, when you have the science and you have the strategy to manage cancer effectively with minimal toxicity, not to say we can cure, but to say we can manage it, why is it not being done? That's the question. But to them, to them who've changed in. Okay. So these kinds of conversations that we have are allowing the populations to realize that there are loved ones do not need to be sacrificed for the good of industries that are generally considered profitable. In other words, the profitability of the industries are based on your sickness. And a lot of those comments came, "Oh, you can't do anything." "Yikes, you can do something about it." When you're armed with the knowledge and people ignore the knowledge, then there's a problem. Do people need to sort of self-advocate to some degree? I think so. With that, with care providers, what do they do? Yeah, I think that's a very delicate question. The oncologist never heard of this stuff. They have never read these papers. They were never trained in medical school to know the biology and biochemistry of cancer. It was told to be a genetic disease. Theories are so important in science. For 1,800 years, people thought the work of Aristotle, his comments, and the mathematics of Claudius Toulami said that the Earth was the center of the solar system and all the planets and sun revolved around the Earth, the geocentric theory, right? Copernicus struggled with the Toulami mathematics and realized that if he put the sun in the center of the solar system and made Earth just another planet, a lot of the mathematics made sense. Kepler comes in and says, these aren't circles, they're ellipticals. Galileo takes the telescope, sees the moons of Jupiter and was able to look and quantify where predict where planets would be in a certain period of time. Then they took poor Gidano Bruno. You know about this guy? Bruno? Stephen, you've got to know Bruno. Your job is to know about the poor Bruno, who was burned alive by the Catholic Church for challenging the geocentric theory. And he became a martyr of science. So when you have established power structure, whether it's a religion or whether it's an industry or whatever, challenging that can be very, very hazardous. Has it been hazardous for you? Listen, no, I mean, I do what I do because I like, I just call it more and more data to support. Hazardous for me would be getting blindsided. Blindsided would be somebody coming at me with a piece of new data that I have never considered. I don't sleep. I think about this stuff all the time to avoid the blind side. My students are on the alert for any paper that comes out that says cancer, oh, cancer cells can burn fatty acids and ketone bodies. Oh, really? Let's go back through and dissect out their control experiments and you find that every case there was always some glucose and glutamine in the media, making it look like the fatty acids. So that's what bothers me. What bothers me is getting hit with a piece of data that undermines what we are knowledge base is. And so far, we haven't had that. We're standing on the shoulders of Otto Warberg, a giant in the field of biochemistry. He was thrown under the bus when everybody thought cancer was a genetic disease. My paper goes back and shows exactly where Warberg made his mistakes and where we have rectified some of that, bringing the whole field back on where it should be. It is a mitochondrial metabolic disorder. And we can account for all of the phenotypes and characteristics of that disease knowing that. Now with that knowledge, logical people and people interested in helping others will take advantage of that. We're not throwing out all these toxic chemicals. We're learning how to use them in a different way. And that's where the success is going to come. So let's conclude with a actionable takeaway for people who are suffering themselves with cancers or have someone in their family right now that is suffering from cancers. What is the actionable takeaway? Well, I think the action will take and once this paper comes out, they can start taking action if they are motivated enough. They can read about this. They can read about it and then try to, like just like you're doing, no different. Get into these zones and then work with their oncologists, knowledgeable people to treat them with standards of care as long as they can remain. And then we do non-invasive imaging, PET scan, MRIs. Okay, so specifically what you're saying is this paper, I will link it below in the comments section for anyone that wants to read it. This graph will be on the screen throughout the subset anyway so people can screenshot it if they want to look. And the way that they test what their GKI index is, is they can buy one of these keto mojo things which you can get an Amazon for $20, $30. You prick your finger, it gives you the glucose reading. You divide it by 18. No, no, the new machines have the button. So even the people only have to do that. Okay, fine. And as you can see on here, this is an interesting way to sort of increase your management, improve your management of some of these. Yeah, and then there's a challenge to get into those zones. I'm not saying this is easy stuff. GLP one inhibitor, man, that's a hell of a lot easier than doing this. And I should probably say always consult with a medical professional. Yeah, I think because you know, a lot of people, they have a lot of colomorbidities. They have diabetes, high blood pressure, hypertension, cancer. They have a, it's not a perfectly healthy person with cancer. So before you go into challenge that, you need to have what you look like. You might, you know, you might have to be adjusted in some way. That's why the people who, the physicians that are working with this can do all that. I'm not in the clinical thing. And there are some people who respond poorly to the ketogenic diet as the high-states ketosis. 'Cause I've received the M's before a woman who said, my husband did ketosis and he collapsed unconscious. Took him to the hospital, he had some comorbidity. Yeah, yeah, yeah. The other thing too is you gotta be, because some people have carnitine deficiencies. Carnitine prevents fatty acids from being made into ketone bodies. So they can be carnitine supplementation can help them. But you need a physician to know this. We have a closing tradition where the last question is lead to questions for the next. And a question left for you is on the subject of energy. What in your life? has brought you the most energy and what was the biggest energy drain you've ever experienced? This is bringing me the biggest energy. Well, that's the whole concept. The idea that you have found Mother Nature has allowed you to look into the depths of what we consider the biology and biochemistry of how bodies work and knowing how to take that and apply it to people that are suffering from all these different chronic diseases and giving them the opportunity to change that because before that it was mysterious. Oh, I'm doing I'm in Kito. What's your GK? I don't know. Well, now you have a quantitative opportunity. That gets us. So we have a lot of evidence. I think people should be feeling encouraged. I think we have given hope to the hopeless. And I think that's empowering. And the goal here is not to make a billion dollars. It's just to know that you've kept all these poor souls alive longer than they were projected to be. To me, and I think this power and that keeps us going because when we see more and more people coming to me, I get emails back from people three or four years ago and I said, "You know, I thought you were a goner." And he said, "Oh, I'm doing really well. Just came back from a vacation with my wife." Well, that's empowering. I said, "Well, that's good." And don't forget, Steven, all of our research money comes from private foundations and full answer, Pete. So there's that way that people can help? Yeah. And where do they go? Travis Christopher since foundation. So we in my papers, we have the foundations that support our work, private foundations. So there are occasionally, yes, please link. There are occasionally people. I know when I give kits of information to people, I say, "Please consider making a donation only if it works for you. Don't charge them anything or ask them to pay something if it's not going to help them. If you were told to be dead in six months and six years later you're alive, maybe throw us a few shuckles into the private foundations supporting our work." I'll link that foundation below in the comments. We have a couple on breast cancer, on general support, on the metabolic approach that we have. So we have a lot going. We're very excited. You can see all the papers we've published. It's not like some of these are in top journals. Some of them are in new journals. But the issue is we're publishing this. My thing is, I have great respect for science and doctors in the medical profession. Go get your information, speak to your medical provider. There's so many tools out there now. Go and check for yourself. Yeah, well, I think in the oncology field, that's where we have this vast wasteland of misinformation or misunderstanding. Let's put it that way. They don't understand these concepts where people who have done hard work and bone work and replacements, those people are at the state of the art with this kind of stuff. Well, the last conversation we had is reached about 15 million people. So on YouTube, it's got 10 million views and then across audio platforms it's got another 5 or 7 million views. When I read through the comment sections, it's just so heartening. For two reasons. One is, you're giving people tools and science that they can think about and research themselves and speak to their medical practitioner about. But also, it actually just creates a community of people in the comment section where this is a community of people that are searching for hope. Yeah. And as some of them read through the comment section, it's actually commented saying it was so nice to speak to other people in the comment sections about what I'm going through and how it feels from an emotional level. So this is something I actually wanted to say in this episode was, if you're listening to this conversation now and you've got into this point, do you feel free to go into the comment section and just offer some support and some love to other people who are struggling because it can be a very lonely experience. The minute you find out, you've got a diagnosis and off you go into the internet, into podcasts, into AI to try and figure out what you can do. So yeah, do share things that have helped you point at different resources that are rigorous and offer emotional support to those there in the comment section. That would be a wonderful thing. Thomas, thank you so much for all that you do. You're an absolute warrior for pushing science into the world and for fighting for these people that don't have the tools. And if you I mean, I don't think I've ever seen a comment section quite like it in terms of the gratitude that people have for the work that you're doing. It is remarkable work. Long may you continue to do it. Thank you very much. And you know, you play a very important part on this because this information is not disseminated to the population. And it's the population of people that will eventually make the change. So they're going to want this, especially when we keep publishing more and more case reports of successful cases. And the change, the system will change. And we just have to modify what we have to make it better. And I think that's what keeps us going. So I have no plans of stopping this anytime soon. My students are all excited about this. They're learning about it at Boston College. So this is a big emphasis that we have. And we continue to do it again. Scientific literacy is so important for how you navigate through life. And thank you very much for your show. And we'll keep pushing this as hard as we can. Thank you. # Few days before the show might start to do theirelnörung when they require measures or design wrong along their way. <i>! Showancy was one of the novelest 중요한 installments where I just placed no blame on the topic. We are also going to show you the shop. Let's start with the shop-finding shop.com.com.